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However, due to the lack of IF in patients with PA, the intrinsic-factor-mediated absorption is disrupted and the efficiency by which Cbl is absorbed by the ileum is reduced to 1-5% via passive diffusion

Although additional studies are required to clarify the molecular mechanisms responsible for hyperhomocysteinemia-induced vascular disorders, there is evidence that the direct cytotoxicity of markedly elevated homocysteine and/or of its oxidized by-products (homocysteine thiolactone, homocysteine sulfinic acid, and homocysteic acid) damages endothelial cells (Dudman et al, 1991

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